![]() However, the mechanisms for its action, and ultimately for resistance, have not been fully defined. Nature Communications volume 10, Article number: 2400 ( 2019)īET-bromodomain inhibition (BETi) has shown pre-clinical promise for MYC-amplified medulloblastoma. Neuronal differentiation and cell-cycle programs mediate response to BET-bromodomain inhibition in MYC-driven medulloblastoma
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